Raw milk accelerates gentle exfoliation and fades surface-level pigmentation, while aloe vera with its lignins penetrate deeper layers, targeting internal inflammation and preventing post-inflammatory marks. Saunf (fennel) inhibit melanin overproduction and oxidative stress, making skin appear visibly clearer and more even-toned.

Aloe vera cools and repairs photodamage, while saunf contains natural SPF-boosting compounds that reduce UV-induced pigmentation. Sesame oil’s vitamin E and zinc reinforce the skin’s antioxidant defence system and reduce redness post-sun exposure, not only reducing existing tan but also strengthening the skin's resistance to future sun damage.

Sunflower seed oil protects lipid membranes from oxidative degradation, while shea butter promotes fibroblast stimulation and prevents collagen breakdown. Raw milk further promotes cellular renewal to slow the onset of visible ageing and preserve skin texture.

Sesame oil and sunflower seed oil with rich fatty acids reinforce elasticity and prevent trans-epidermal water loss. Aloe's mucopolysaccharides and shea maintain structural bounce and suppleness even in deeper skin layers.

Saunf offers vitamin C and bioactive polyphenols that protect fibroblasts from oxidative damage and stimulate pro-collagen production. Shea butter supports this process by nourishing the dermis to promote repair.

Shea butter and sesame oil both soothe itch and reduce inflammation-driven flare-ups. Aloe vera’s polysaccharides further promote calm and restore skin tolerance. Raw milk’s natural fats and proteins help rebuild the hydrolipid layer, strengthening barrier function against external allergens and chemical irritants.

Malai offers light-reflective hydration without heaviness, while raw milk enzymes smoothen skin texture. Aloe and fennel’s antioxidants help neutralise free radicals that cause dullness. Sesame and shea oils restore skin lipids, locking in a moisturised glow. Together, they leave skin with a sheen that signals vitality, not greasiness.

Glycerin often gives the illusion of moisturised skin because it makes the surface feel soft and supple immediately after application. However, this is often superficial and short-lived. In reality, it may leave the deeper skin layers more vulnerable.

While glycerin is a powerful humectant that attracts water, in low-humidity environments or when applied without sufficient occlusion, it can pull moisture and lipids out from the deeper layers of the skin and from surrounding intercellular spaces. This can compromise the skin's lipid matrix—especially ceramides, cholesterol, and fatty acids—leading to a weakened skin barrier.

Ironically, though glycerin is used to hydrate, it does only surface hydration while simultaneously dehydrating the deeper layers. Without a protective occlusive layer to seal in moisture, skin can become dependent on external hydration, leading to rebound dryness and dehydration beneath the surface.

Chronic dehydration and lipid depletion caused by glycerin can reduce the skin’s natural elasticity. Over time, this compromises skin firmness and may contribute to the appearance of fine lines, sagging, and loss of structural resilience—hallmarks of premature aging.

A compromised barrier and dehydrated dermis create oxidative stress and inflammatory microenvironments in the skin. These conditions can disrupt fibroblast activity and collagen synthesis while accelerating collagen breakdown, indirectly weakening the structural scaffolding of the skin.

The skin’s hydrolipid film is a delicate emulsion of sebum, sweat, and lipids that coats the epidermis. When this barrier is intact, it holds moisture and prevents the penetration of pollutants and UV-induced oxidative agents. However, exposure to sun, wind, and detergents can strip this layer, leading to dryness, inflammation, and compromised UV resilience.

Hyaluronic acid (HA) and natural moisturizing factors (NMFs) reside within the epidermis and bind water molecules to retain hydration. These molecules not only plump and smooth the skin but also help create a cellular environment where DNA repair and antioxidant activity function optimally. Dehydrated skin, especially on the body, becomes more photosensitive, more prone to UV-induced enzyme breakdown, and less capable of bouncing back from sun exposure.

Ultraviolet radiation penetrates deep into the dermis and initiates free radical formation, which disrupts cellular lipids, impairs aquaporins (water channels in skin cells), and accelerates transepidermal water loss. This explains why sun-exposed areas like the arms and shoulders often feel parched, tight, and leathery post-exposure. Prolonged UV damage also reduces the skin’s ability to synthesize its own ceramides, making it increasingly dry over time.

Topical antioxidants like vitamin C, vitamin E, & polyphenols don’t just protect against UV damage; they also stabilize and enhance the skin’s water-retention ability. They neutralise reactive oxygen species (ROS) generated by sun exposure, preventing lipid peroxidation and protein breakdown in the barrier.

Even with protection, sub-clinical UV damage occurs after sun exposure, this includes micro-inflammation, dehydration, and lipid oxidation. Post-sun care should go beyond cooling and aim to actively rehydrate and repair. Ingredients like aloe vera not only replenish lost water but also support cellular repair pathways and soothe sun-induced irritation.